Traumatic Rupture of the Urinary Tract in a Patient Presenting Nephrogenic Diabetes Insipidus Associated with Hydronephrosis and Chronic Renal Failure: Case Report and Review of the Literature
| Title: | Traumatic Rupture of the Urinary Tract in a Patient Presenting Nephrogenic Diabetes Insipidus Associated with Hydronephrosis and Chronic Renal Failure: Case Report and Review of the Literature |
|---|---|
| Authors: | Zender, H.O.; Ruedin, P.; Moser, F.; Bolle, J.F.; Leski, M. |
| Publisher: | Clinical Nephrology |
| Date Published: | October 01, 1992 |
| Reference Number: | 297 |
We report the case of a 34-year-old Japanese man suffering from a nephrogenic diabetes insipidus (NDI) associated with bilateral hydronephrosis, hydroureters and enlarged trabeculated bladder without obstruction. He also presented with chronic renal failure which has rarely occurred in similar cases. The patient was admitted after a traumatic rupture of the left urinary tract which had never been described until now in NDI. He was treated successfully by transient peritoneal and vesical drainages. This paper focuses on the very rare complication of chronic renal failure secondary to hydronephrosis in cases of NDI. The literature of this association is reviewed.
This translation by the NDI Foundation is to assist the lay reader. To provide a clear, accessible interpretation of the original article, we eliminated or simplified some technical detail and complicated scientific language. We concentrated our translation on those aspects of the article dealing directly with NDI. The NDI Foundation thanks the researchers for their work toward understanding and more effectively treating this disorder.
© Copyright NDI Foundation 2007 (JC)
The authors inserted a catheter into the patient's bladder for 10 days and a catheter into the membrane lining the abdomen for 24 hours. This eliminated the patient's pain, though it did not treat his NDI. Zender, et al's, patient is the 33rd case of NDI associated with distention of kidney pelvises and calices reported in the literature. Many researchers believe bladder enlargement can be due to the chronic, excessive passage of large volumes of urine resulting from NDI. This can be aggravated if the NDI patient habitually retains urine instead of voiding it when the urge arises. Most of the patients suffering from NDI with associated hydronephrosis did not undergo operations to treat this condition.
The authors' patient also presented with chronic kidney failure caused by the severity of his hydronephrosis, which thinned his kidney cortex. The authors emphasize that preventative care is mandatory for NDI patients in order to prevent hydronephroses and the risk of associated kidney failure. Preventative care should include early medical treatment and frequent urination.