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2004 - Phoenix
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Sei Sasaki
Title:
M.D.
Email:
Phone:
Work +81 358-035212, Fax +81 358-030132
Organization:
Tokyo Medical & Dental University
Department:
Homeostasis Medicine and Nephrology
Address:
1-5-45 Yushima, Bunkyo-KU
Tokyo, 113-8519
Japan
28 Articles
28 Articles
The role of actin remodeling in the trafficking of intracellular vesicles, transporters, and channels: focusing on aquaporin-2
Pathogenesis and Treatment of Autosomal-Dominant Nephrogenic Diabetes Insipidus Caused by an Aquaporin 2 Mutation
The C-Terminal Tail of Aquaporin-2 Determines Apical Trafficking
Identification of a Multiprotein "Motor" Complex Binding to Water Channel Aquaporin-2
Hypertonicity Regulates the Aquaporin-2 Promoter Independently of Arginine Vasopressin
Molecular Mechanisms and Drug Development in Aquaporin Water Channel Diseases: Molecular Mechanism of Water Channel Aquaporin-2 Trafficking
Water Channel Aquaporin-2 Directly Binds to Actin
Aquaporin-2 Trafficking is Regulated by PDZ-domain Containing Protein SPA-1
Clinical Characteristics of Eight Patients with Congenital Nephrogenic Diabetes Insipidus
Nephrogenic Diabetes Insipidus: Update of Genetic and Clinical Aspects
A Case of a Novel Mutant Vasopressin Receptor-Dependent Nephrogenic Diabetes Insipidus With Bilateral Non-Obstructive Hydronephrosis in a Middle Aged Man
Vasopressin-Independent Renal Urinary Concentration: Increased rBSC1 and Enhanced Countercurrent Multiplication
Pathogenesis of Nephrogenic Diabetes Insipidus by Aquaporin-2 C-Terminus Mutations
Reverse Pharmacological Effect of Loop Diuretics and Altered rBSC1 Expression in Rats with Lithium Nephropathy
Successful Treatment of Partial Nephrogenic Diabetes insipidus with Thiazide and Desmopressin
Two Novel Aquaporin-2 Mutations Responsible for Congenital Nephrogenic Diabetes Insipidus in Chinese Families
Water Intake and 24-hour Blood Pressure Monitoring in a Patient with Nephrogenic Diabetes Insipidus Caused by a Novel Mutation of the Vasopressin V2R Gene [Letter; Comment]
Three Families with Autosomal Dominant Nephrogenic Diabetes Insipidus Caused by Aquaporin-2 Mutations in the C-terminus
Impaired Solute Accumulation in Inner Medulla of Clcnk1-/- Mice Kidney
Report of 33 Novel AVPR2 Mutations and Analysis of 117 Families with X-Linked Nephrogenic Diabetes Insipidus
Functional Analysis of Aquaporin-2 Mutants Associated with Nephrogenic Diabetes Insipidus by Yeast Expression
Overt Nephrogenic Diabetes Insipidus in Mice Lacking the CLC-K1 Chloride Channel
Novel Mutations in Aquaporin-2 Gene in Female Siblings with Nephrogenic Diabetes Insipidus: Evidence of Disrupted Water Channel Function
Urinary Excretion of Aquaporin-2 in Disorders of Water Metabolism
Urinary Excretion of Aquaporin-2 in Patients with Diabetes Insipidus
Isolation of Human aquaporin-CD Gene
Cloning, Characterization, and Chromosomal Mapping of Human Aquaporin of Collecting Duct
Cloning and Expression of Apical Membrane Water Channel of Rat Kidney Collecting Tubule
5 Conference Proceedings
5 Conference Proceedings
Nephrogenic Diabetes Insipidus Patients in Japan
Contribution of chloride channels in urine concentrating ability
Gene Mutation Analysis of NDI patients in Japan
Expressional alteration of sodium transporter in the thick ascending limb of Henle and urinary concentration
Clinical analysis of eight Japanese patients with congenital nephrogenic diabetes insipidus