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2004 - Phoenix
2002 - Heemskerk
2000 - La Jolla
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1998 - La Jolla
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Chung-Lin Chou
Title:
Staff Scientist
Email:
Phone:
Work (301) 496-3636, Fax (301) 402-1443
Organization:
National Heart, Lung and Blood Institute
Division:
National Institutes of Health
Department:
Laboratory of Kidney and Electrolyte Metabolism
Address:
Bldg. 10, Rm. 6N260 10 Center Drive MSC 1603
Bethesda, MD 20892-1603
United States
17 Articles
17 Articles
Nonmuscle Myosin II and Myosin Light Chain Kinase are Downstream Targets for Vasopressin Signaling in the Renal Collecting Duct
Application of Difference Gel Electrophoresis (DIGE) to the Identification of Inner Medullary Collecting Duct Proteins
Proteomic Analysis of Long-Term Vasopressin Action in Inner Medullary Collecting Duct of Brattleboro Rat
Regulation of Aquaporin-2 Trafficking by Vasopressin in the Renal Collecting Duct
Escape from Vasopressin-Induced Antidiuresis: Role of Vasopressin Resistance of the Collecting Duct
Fourfold Reduction of Water Permeability in Inner Medullary Collecting Duct of Aquaporin-4 Knockout Mice
Syntaxin-4 is Localized to the Apical Plasma Membrane of Rat Renal Collecting Duct Cells: Possible Role in Aquaporin-2 Trafficking
Renal Aquaporins
Evidence for Dual Signaling Pathways for V2 Vasopressin Receptor in Rat Inner Medullary Collecting Duct
Extracellular Nucleotide Receptor Inhibits AVP-Stimulated Water Permeability in Inner Medullary Collecting Duct
Oxytocin as an Antidiuretic Hormone. II. Role of V2 Vasopressin Receptor
Vasopressin Increases Water Permeability of Kidney Collecting Duct by Inducing Translocation of Aquaporin-CD Water Channels to Plasma Membrane
Mechanism of Vasopressin Action in the Renal Collecting Duct
Urinary Concentrating Mechanism: The Role of the Inner Medulla
Experimental Tests of Three-Dimensional Model of Urinary Concentrating Mechanism
Kinetics of Urea and Water Permeability Activation by Vasopressin in Rat Terminal IMCD
Regulation of Collecting Duct Water Permeability Independent of cAMP-Mediated AVP Response
2 Conference Proceedings
2 Conference Proceedings
Urinary Concentrating Defect in Mice With Selective Deletion of Collecting-Duct Urea Transporter Isoforms, UT-A1 and UT-A3
Altered expression pattern of solute transporters in inner medulla of AQP1 (-/-) and CLC-nK1 (-/-) mice