American Journal of Physiology: Renal Physiology
41 Articles
- Altered Expression of COX-1, COX-2, and mPGES in Rats with Nephrogenic and Central Diabetes Insipidus
- Altered Expression of Major Renal Na Transporters in Rats with Unilateral Ureteral Obstruction
- Altered Expression of Renal AQPs and Na+ Transporters in Rats with Lithium-Induced NDI
- Angiotensin II AT1 Receptor Blockade Decreases Vasopressin-Induced Water Reabsorption and AQP2 Levels in NaCl-Restricted Rats
- Application of Difference Gel Electrophoresis (DIGE) to the Identification of Inner Medullary Collecting Duct Proteins
- AQP3, p-AQP2, and AQP2 Expression is Reduced in Polyuric Rats With Hypercalcemia: Prevention by cAMP-PDE Inhibitors
- Aquaporin-2: COOH Terminus is Necessary But Not Sufficient for Routing to the Apical Membrane
- Bidirectional Regulation of AQP2 Trafficking and Recycling: Involvement of AQP2-S256 Phosphorylation
- cDNA Array Identification of Genes Regulated in Rat Renal Medulla in Response to Vasopressin Infusion
- Changes in Renal Medullary Transport Proteins During Uncontrolled Diabetes Mellitus in Rats
- Chlorpropamide Upregulates Antidiuretic Hormone Receptors and Unmasks Constitutive Receptor Signaling
- Concentration of Solutes in the Renal Inner Medulla: Interstitial Hyaluronan as a Mechano-Osmotic Transducer
- Development of Water Transport in the Collecting Duct
- Downregulation of AQP1, -2, and -3 After Ureteral Obstruction is Associated With a Long-Term Urine-Concentrating Defect
- Downregulation of Aquaporin-2 and -3 in Aging Kidney is Independent of V2 Vasopressin Receptor
- Downregulation of Renal Aquaporins in Response to Unilateral Ureteral Obstruction
- Downregulation of Renal Vasopressin V2 Receptor and Aquaporin-2 Expression Parallels Age-Associated Defects in Urine Concentration
- Effect of Water Deprivation and Hypertonic Saline Infusion on Urinary AQP2 Excretion in Healthy Humans
- Effects of Dietary Fat, NaCl, and Fructose on Renal Sodium and Water Transporter Abundances and Systemic Blood Pressure
- Evidence for a Role of Protein Kinase C-a in Urine Concentration
- Impaired Solute Accumulation in Inner Medulla of Clcnk1-/- Mice Kidney
- Importance of Aquaporin-2 Expression Levels in Genotype -Phenotype Studies in Nephrogenic Diabetes Insipidus
- In Vivo Role of CLC Chloride Channels in the Kidney
- Inhibition of Endocytosis Causes Phosphorylation (S256)-Independent Plasma Membrane Accumulation of AQP-2
- Insulin Potentiates AVP-induced AQP2 Expression in Cultured Renal Collecting Duct Principal Cells
- N-ethylmaleimide (NEM) Causes Aquaporin-2 Trafficking in the Renal Inner Medullary Collecting Duct by Direct Activation of Protein Kinase A
- Osmolality and Solute Composition are Strong Regulators of AQP2 Expression in Renal Principal Cells
- Proteomic Analysis of Long-Term Vasopressin Action in Inner Medullary Collecting Duct of Brattleboro Rat
- Renal Expression of Sodium Transporters and Aquaporin-2 in Hypothyroid Rats
- Rho Inhibits cAMP-Induced Translocation of Aquaporin-2 into the Apical Membrane of Renal Cells
- Segment Specific ENaC Downregulation in Kidney of Rats with Lithium-Induced NDI
- Structure and Function of Aquaporin Water Channels
- The Ins and Outs of Aquaporin-2 Trafficking
- Stimulation of AQP2 Membrane Insertion in Renal Epithelial Cells In Vitro and In Vivo by the cGMP Phosphodiesterase Inhibitor Sildenafil Citrate (Viagra)
- Characterization of Vasopressin V2 Receptor Mutants in Nephrogenic Diabetes Insipidus in a Polarized Cell Model
- Genetic Restoration of Aldose Reductase to the Collecting Tubules Restores Maturation of the Urine Concentrating Mechanism
- Methyl-β-Cyclodextrin Induces Vasopressin-Independent Apical Accumulation of Aquaporin-2 in the Isolated, Perfused Rat Kidney
- Functional Rescue of Vasopressin V2 Receptor Mutants in MDCK Cells by Pharmacochaperones: Relevance to Therapy of Nephrogenic Diabetes Insipidus
- Urea Transporter UT-A1 and Aquaporin-2 Proteins Decrease in Response to Angiotensin II or Norepinephrine-Induced Acute Hypertension
- Amiloride restores renal medullary osmolytes in lithium-induced nephrogenic diabetes insipidus
- Treating lithium-induced nephrogenic diabetes insipidus with a COX-2 inhibitor improves polyuria via upregulation of AQP2 and NKCC2.