collecting duct
The definitions used in this glossary of terminology either have been provided by the authors of the articles, or have been extracted wholly or in part, or paraphrased from the following sources: The American Medical Association Encyclopedia of Medicine, Charles B. Clayman, MD, Medical Editor, Random House, New York, 1989; Biotechnology from A to Z, 2d Edition, William Bains, Oxford University Press, New York, New York, 2002; A Dictionary of Genetics, 6th Edition, Robert C. King and William D. Stansfield, Oxford University Press, New York, New York, 2002; Dorland's Illustrated Medical Dictionary, 29th and 30th Editions, W. B. Saunders Company, Philadelphia, 2000, 2003; Genes VII, Benjamin Lewin, Oxford University Press, New York, New York, 2000; The Gale Encyclopedia of Genetic Disorders, Volumes I and II, Stacey L. Blachford, Ed., Thomson Learning, New York, New York, 2002; The Merriam-Webster Dictionary, Merriam-Webster, Inc., Springfield, Massachusetts, 1997; Molecular Biology of the Cell, 3rd Edition, Bruce Alberts, et al., Garland Publishing, 1994; The Random House Dictionary of the English Language, Unabridged Edition, 1966; Webster's Ninth New Collegiate Dictionary, 1991.
DEFINITION:
- collecting duct
-
See under duct.
Used in 68 Article abstracts
Used in 68 Article abstracts
- A Case of Nephrogenic Diabetes Insipidus Caused by Obstructive Uropathy Due to Prostate Cancer
- AQP3, p-AQP2, and AQP2 Expression is Reduced in Polyuric Rats With Hypercalcemia: Prevention by cAMP-PDE Inhibitors
- Amelioration of Polyuria in Nephrogenic Diabetes Insipidus Due to Aquaporin-2 Deficiency
- Aquaporin-2 Water Channel Mutations Causing Nephrogenic Diabetes Insipidus
- Aquaporins in the Kidney: From Molecules to Medicine
- Aquaporins: From Physiology to Nephrogenic Diabetes Insipidus
- Bilateral Ureteral Obstruction Downregulates Expression of Vasopressin-Sensitive AQP-2 Water Channel in Rat Kidney
- Bradykinin Signaling Counteracts cAMP-elicited aquaporin 2 translocation in renal cells
- Changes of Rat Kidney AQP2 and Na,K-ATPase mRNA Expression in Lithium-Induced Nephrogenic Diabetes Insipidus
- Characterization of Purified Endosomes Containing the Antidiuretic Hormone-Sensitive Water Channel From Rat Renal Papilla
- Cloning, Characterization, and Chromosomal Mapping of Human Aquaporin of Collecting Duct
- Concentrating Defect in Experimental Nephrotic Syndrome: Altered Expression of Aquaporins and Thick Ascending Limb Na+ Transporters
- Dehydration Reverses Vasopressin Antagonist-Induced Diuresis and Aquaporin-2 Downregulation in Rats
- Development of Urinary Concentrating Capacity: Role of Aquaporin-2
- Downregulation of AQP1, -2, and -3 After Ureteral Obstruction is Associated With a Long-Term Urine-Concentrating Defect
- Downregulation of Aquaporin-2 Parallels Changes in Renal Water Excretion in Unilateral Ureteral Obstruction
- Dynein and dynactin colocalize with AQP2 water channels in intracellular vesicles from kidney collecting duct
- Endosomes from Kidney Collecting Tubule Cells Contain the Vasopressin-sensitive Water Channel
- Escape from Vasopressin-Induced Antidiuresis: Role of Vasopressin Resistance of the Collecting Duct
- Evidence for Dual Signaling Pathways for V2 Vasopressin Receptor in Rat Inner Medullary Collecting Duct
- Evidence for Stabilization of Aquaporin-2 Folding Mutants by N-linked Glycosylation in the Endoplasmic Reticulum
- Experimental Tests of Three-Dimensional Model of Urinary Concentrating Mechanism
- Expression of Renal Aquaporins 1, 2, and 3 in a Rat Model of Cisplatin-Induced Polyuria
- Expression of VAMP-2-Like Protein in Kidney Collecting Duct Intracellular Vesicles. Colocalization with Aquaporin-2 Water Channels
- Foscarnet Alters Antidiuretic Hormone-Mediated Transport
- Impaired Aquaporin and Urea Transporter Expression in Rats with Adriamycin-Induced Nephrotic Syndrome
- Increased Renal Responsiveness to Vasopressin and Enhanced V2 Receptor Signaling in RGS2-/- Mice
- Indomethacin Enhances Shuttling of Aquaporin-2 Despite Decreased Abundance in Rat Kidney
- Intrafamilial Phenotype Variability in Nephrogenic Diabetes Insipidus
- Long Term Regulation of Aquaporin-2 Expression in Vasopressin-responsive Renal Collecting Duct Principal Cells
- Long-Term Regulation of Four Renal Aquaporins in Rats
- Long-term Regulation of Aquaporins in the Kidney
- Loss of Calcineurin Aα Results in Altered Trafficking of AQP2 and in Nephrogenic Diabetes Insipidus
- Methyl-β-Cyclodextrin Induces Vasopressin-Independent Apical Accumulation of Aquaporin-2 in the Isolated, Perfused Rat Kidney
- Molecular Pathogenesis of Nephrogenic Diabetes Insipidus
- Molecular Physiology of Urinary Concentrating Mechanism: Regulation of Aquaporin Water Channels by Vasopressin
- Nephrogenic Diabetes Insipidus in Sibling Colts
- Oxytocin Induces Apical and Basolateral Redistribution of Aquaporin-2 in Rat Kidney
- Pathophysiology of Aquaporin-2 in Water Balance Disorders
- Pathophysiology of Renal Fluid Retention
- Pathophysiology of the Aquaporin Water Channels
- Physiology and Pathophysiology of Renal Aquaporins
- Proteomic Analysis of Long-Term Vasopressin Action in Inner Medullary Collecting Duct of Brattleboro Rat
- Rat Kidney Papilla Contains Abundant Synaptobrevin Protein that Participates in the Fusion of Antidiuretic Hormone-regulated Water Channel-containing Endosomes In Vitro
- Recent Advances in Water Transport
- Regional Time-Dependent Changes in Vasopressin V2 Receptor Expression in the Rat Kidney During Water Restriction
- Regulation of Aquaporin-2 Water Channel Trafficking by Vasopressin
- Regulation of Collecting Duct Water Channel Expression by Vasopressin in Brattleboro Rat
- Renal Aquaporins
- Reverse Pharmacological Effect of Loop Diuretics and Altered rBSC1 Expression in Rats with Lithium Nephropathy
- Role of Aquaporin Water Channels in Kidney and Lung
- Role of Aquaporins in Water Balance Disorders
- Role of Inner Medullary Collecting Duct NaCl Transport in Urinary Concentration
- Role of the Ca2+-Sensing Receptor in Divalent Mineral Ion Homeostasis
- Segment Specific ENaC Downregulation in Kidney of Rats with Lithium-Induced NDI
- Structure and Function of Kidney Water Channels
- Syntaxin-4 is Localized to the Apical Plasma Membrane of Rat Renal Collecting Duct Cells: Possible Role in Aquaporin-2 Trafficking
- The Molecular Structure of the Antidiuretic Hormone Elicited Water Channel
- Tonicity-Responsive Enhancer Binding Protein is an Essential Regulator of Aquaporin-2 Expression in Renal Collecting Duct Principal Cells
- Transport Defects of Rabbit Inner Medullary Collecting Duct Cells in Obstructive Nephropathy
- Treating lithium-induced nephrogenic diabetes insipidus with a COX-2 inhibitor improves polyuria via upregulation of AQP2 and NKCC2.
- Upregulation of Collecting Duct Aquaporin-2 by Metabolic Acidosis: Role of Vasopressin
- Urinary Aquaporin-2 Excretion in Dogs: A Marker for Collecting Duct Responsiveness to Vasopressin
- Vasopressin Increases Water Permeability of Kidney Collecting Duct by Inducing Translocation of Aquaporin-CD Water Channels to Plasma Membrane
- Vasopressin Receptors in Health and Disease
- Water Channels Encoded by Mutant Aquaporin-2 Genes in Nephrogenic Diabetes Insipidus are Impaired in Their Cellular Routing
- Water Disturbances in Cardiac Failure
- [Nephrogenic Diabetes Insipidus] (Japanese)
Used in 5 Article bodies
Used in 5 Article bodies
- Hyperosmolar Coma and Lithium-induced Diabetes Insipidus
- Nephrogenic Diabetes Insipidus
- Nephrogenic Diabetes Insipidus: A Cause of Severe Nonobstructive Urinary Tract Dilatation
- Role of Aquaporin-2 Water Channels in Urinary Concentration and Dilution Defects
- Role of Vasopressin in Abnormal Water Excretion in Cirrhotic Patients
Used in 54 Article translations
Used in 54 Article translations
- A Heterotrimeric G Protein of the Gi Family is Required for cAMP-triggered Trafficking of Aquaporin 2 in Kidney Epithelial Cells
- A Variant of Nephrogenic Diabetes Insipidus: V2 Receptor Abnormality Restricted to the Kidney
- ADH Resistance of LLC-pk1 Cells Caused by Overexpression of cAMP-Phosphodiesterase Type-IV
- Activation of the Vasopressin-sensitive Water Permeability Pathway in the Toad Bladder by N-ethyl Maleimide
- Altered Expression of COX-1, COX-2, and mPGES in Rats with Nephrogenic and Central Diabetes Insipidus
- Assignment of the Human Gene for the Water Channel of Renal Collecting Duct Aquaporin 2 (AQP2) to Chromosome 12 Region q12-->q13
- Bilateral Ureteral Obstruction Downregulates Expression of Vasopressin-Sensitive AQP-2 Water Channel in Rat Kidney
- Cellular Distribution of the Aquaporins: A Family of Water Channel Proteins
- Cellular Mechanism of Lithium-Induced Nephrogenic Diabetes Insipidus in Rats
- Concentrating Defect in Experimental Nephrotic Syndrome: Altered Expression of Aquaporins and Thick Ascending Limb Na+ Transporters
- Congestive Heart Failure in Rats is Associated with Increased Expression and Targeting of Aquaporin-2 Water Channel in Collecting Duct
- Cyclooxygenase-2 Inhibitor Preserves Medullary Aquaporin-2 Expression and Prevents Polyuria After Ureteral Obstruction
- Dehydration Reverses Vasopressin Antagonist-Induced Diuresis and Aquaporin-2 Downregulation in Rats
- Development of Lithium-Induced Nephrogenic Diabetes Insipidus is Dissociated from Adenylyl Cyclase Activity
- Discovery of Aquaporins: a Breakthrough in Research on Renal Water Transport
- Do Aquaporins Have a Role in Nocturnal Enuresis?
- Downregulation of Aquaporin-2 Parallels Changes in Renal Water Excretion in Unilateral Ureteral Obstruction
- Dual Actions of Vasopressin and Oxytocin in Regulation of Water Permeability in Terminal Collecting Duct
- Dynein and dynactin colocalize with AQP2 water channels in intracellular vesicles from kidney collecting duct
- Endosomes from Kidney Collecting Tubule Cells Contain the Vasopressin-sensitive Water Channel
- Epinephrine and dDAVP Administration in Patients with Congenital Nephrogenic Diabetes Insipidus. Evidence for a pre-cyclic AMP V2 Receptor Defective Mechanism
- Hyponatremia and Hypernatremia
- Identification of a Novel A-kinase Anchoring Protein 18 Isoform and Evidence for its Role in the Vasopressin-induced Aquaporin-2 Shuttle in Renal Principal Cells
- Impaired Aquaporin and Urea Transporter Expression in Rats with Adriamycin-Induced Nephrotic Syndrome
- Increased Renal Responsiveness to Vasopressin and Enhanced V2 Receptor Signaling in RGS2-/- Mice
- Indomethacin Enhances Shuttling of Aquaporin-2 Despite Decreased Abundance in Rat Kidney
- Indomethacin Treatment in Amphotericin B Induced Nephrogenic Diabetes Insipidus
- Induction of Intramembranous Particle Clusters in Mice with Nephrogenic Diabetes Insipidus
- Mechanism of Lithium-Induced Polyuria in the Rat
- Mechanism of Vasopressin Action in the Renal Collecting Duct
- Mechanisms and Regulation of Water Permeability in Renal Epithelia
- Mechanisms of Impaired Urinary Concentrating Ability in Adult Rats Treated Neonatally with Enalapril
- Mild Nephrogenic Diabetes Insipidus Caused by Foxa1 Deficiency
- Molecular Cloning of the Receptor for Human Antidiuretic Hormone
- Molecular and Cellular Defects in Nephrogenic Diabetes Insipidus
- Nephrogenic Diabetes Insipidus in Sibling Colts
- Osmolality and Solute Composition are Strong Regulators of AQP2 Expression in Renal Principal Cells
- Pathophysiology of the Aquaporin Water Channels
- Platelet Vasopressin Receptors in Patients With Congenital Nephrogenic Diabetes Insipidus
- Properties of the Human Arginine Vasopressin V2 Receptor after Site-Directed Mutagenesis of its Putative Palmitoylation Site
- Relationship Between Vasopressin-Sensitive Water Transport and Plasma Membrane Fluidity in Kidney Collecting Tubule
- Requirement of Human Renal Water Channel Aquaporin-2 for Vasopressin-Dependent Concentration of Urine
- Role of Aquaporin-2 Water Channels in Urinary Concentration and Dilution Defects
- Role of Aquaporins in Water Balance Disorders
- Role of cAMP-Phosphodiesterase Isozymes in Pathogenesis of Murine Nephrogenic Diabetes Insipidus
- Structure and Chromosomal Localization of the Human Antidiuretic Hormone Receptor Gene
- Successful Treatment with Hydrochlorothiazide and Amiloride in an Infant with Congenital Nephrogenic Diabetes Insipidus
- The Aquaporin Family of Water Channel Proteins in Clinical Medicine
- The Aquaporin Family of Water Channels in Kidney: an Update on Physiology and Pathophysiology of Aquaporin-2
- The Cellular Action of Antidiuretic Hormone
- The Clinical Importance of the Urinary Excretion of Aquaporin-2
- Therapeutic Potential of Vasopressin Receptor Antagonists
- Tonicity-Responsive Enhancer Binding Protein is an Essential Regulator of Aquaporin-2 Expression in Renal Collecting Duct Principal Cells
- Urinary Concentrating Mechanism: The Role of the Inner Medulla
Used in 28 Proceeding abstracts
Used in 28 Proceeding abstracts
- Rescue of the Cell Surface Expression of Vasopressin V2 Receptor Mutants in Nephrogenic Diabetes Insipidus
- Amelioration of Polyuria in Nephrogenic Diabetes Insipidus due to Aquaporin-2 Deficiency
- Calcineurin A-alpha knockout is a new model of nephrogenic diabetes insipidus
- Chemical Chaperones as a Novel Therapeutic Strategy for NDI
- Collecting Duct Specific Gene Regulation: Creation And Use of Transgenic Mouse Models
- Correction of age-related polyuria by dDAVP: Molecular involvement of aquaporins and urea transporters
- Expressional alteration of sodium transporter in the thick ascending limb of Henle and urinary concentration
- Gene Mutation Analysis of NDI patients in Japan
- Hormone-regulated intrenalization of the vasopressin V2 receptor in a polarized renal collecting duct cell system
- Identification of proteins involved in the vasopressin-induced shuttle of aquaporin-2
- Long-term regulation of aquaporin-2: a possible therapeutic approach to NDI?
- Modulation of vasopressin-dependent AQP2 expression in cultured mpkCCDc14 collecting duct principal cells by aldosterone, insulin, and hypertonicity
- Nephrogenic Diabetes Insipidus in mice lacking aquaporin-3 water channels
- New insights in water channel physiology revealed by transgenic mouse models
- Obstructive nephropathy -- a common acquired condition of nephrogenic diabetes insipidus
- Osmolality and solute composition regulate aquaporin-2 expression in primary cultured renal principal cells
- Production and purification of Aquaporin-2 and Vasopressin 2 Receptor: towards elucidation of the structure
- Prostaglandins as Stimulants of AQP2 Expression: a Possible Future Treatment for NDI?
- Purinergic Control of Medullary Collecting Duct Function: A Novel Vasopressin-independent Regulatory Mechanism
- Recycling of the V2 Vasopressin Receptor
- Regulation and distribution of phosphorylated aquaporin-2 (AQP2) in rat kidney collecting duct principal cells
- Regulation of AQP2 trafficking and recycling by dopamine, PGE2 and ANP: involvement of AQP2-S256 phosphorylation
- Regulation of renal aquaporins and sodium transporters in conditions with urinary tract obstruction
- The Medical Geneticist's Perspective on NDI Research
- Toward gene therapy for nephrogenic diabetes insipidus
- Urinary Concentrating Defect in Mice With Selective Deletion of Collecting-Duct Urea Transporter Isoforms, UT-A1 and UT-A3
- Vasopressin-Independent Regulation of Aquaporin-2 Protein
- Vasopressin-V2-receptor dependent and independent regulation of collecting duct aquaporin-2 expression and trafficking
Used in 8 Proceeding translations
Used in 8 Proceeding translations
- Long-term regulation of aquaporin-2: a possible therapeutic approach to NDI?
- Obstructive nephropathy -- a common acquired condition of nephrogenic diabetes insipidus
- Prostaglandins as Stimulants of AQP2 Expression: a Possible Future Treatment for NDI?
- Proteomic Studies of Vasopressin Action in the Collecting Duct
- Purinergic Control of Medullary Collecting Duct Function: A Novel Vasopressin-independent Regulatory Mechanism
- Regulation and distribution of phosphorylated aquaporin-2 (AQP2) in rat kidney collecting duct principal cells
- Regulation of Aquaporin-2 trafficking by Vasopressin in Renal Collecting Duct: Roles of Ryanodine-Sensitive Ca2+ Stores and Calmodulin
- The C-terminus of aquaporin-2 is necessary, but not sufficient, for routing of AQP2 to the apical membrane
